We believe in
longevity for all

Genomic Instability

As cells age, their chromosomes become less stable. As repair mechanisms fail to correct DNA damage, mutations accumulate and lead to aging and disease.

Telomere Attrition

As cells divide, telomeres shorten until telomerase, the enzyme that maintains them, is silenced and cell division stops.

Epigenetic Alteration

This type of damage changes how the genome is read, not its DNA sequence, and accumulates as cells are exposed to environmental factors over time.

Loss of Proteostasis

As cells age, environmental stresses cause proteins to lose their stability, autophagic processes to start to fail, and misfolded proteins to accumulate.

Deregulated Nutrient Sensing

Metabolic activities can put stress on our cells. Too much activity and changes in nutrient availability and composition cause cells to age faster.

Mitochondrial Dysfunction

As cells age, oxidative stress damages mitochondrial integrity, compromising function and triggering aging-related events like increased apoptosis.

Cellular Senescence

This process occurs when aging cells lose the ability to divide, stopping growth due to damage or missing components.

Stem Cell Exhaustion

Stem cells lose their ability to divide, and once they migrate, differentiate, or die, they aren’t replaced. Over time, this shows up as outward signs of aging, like gray hair.

Altered Intercellular Communication

As cells age, increased self-preserving signals cause damage elsewhere, and altered communication between cells contributes to declining tissue health.